Talk:Neuropeptide Y

Untitled

Wouldn't it be more appropriate to say that NPY release, rather than production, is influenced in PVN? I thought NPY is produced by cells residing in ARC and released at their terminals in PVN.

Comment on involvement in regulation of Cutaneous Circulation needed in main article

The sympathetic nervous system (SNS) vasoconstrictor innervation has little activity at normal body core temperature. SNS activity goes up in response to body cooling; vasoconstriction occurs via norepinephrine (alpha-adrenergic receptors) and the cotransmitter, neuropeptide Y. Vasoconstriction is necessary during cold temperatures to retain body heat by redirecting the flow of blood from the capillaries directly through Arteriovenous Anastomoses (AVAs), shunting the flow of blood directly from arterioles to venous plexuses. This occurs in acral skin such as in fingers, toes, palms, soles, ears, and the nose.

Amateur question

I'm sure it's not what an expert would ask, but well, if it's 36 amino acids, shouldn't there be a relatively simple way to get from this article to knowing the amino acid sequence? Does the infobox provide a way?

What is it?

ANSWER: try a protein data bank, although it would be nice if there were direct links available on Wiki.

RandomP (talk) 19:21, 25 August 2008 (UTC)

Artificial sweeteners & Neuropeptide Y release

I just heard Jillian Michaels say that artificial sweeteners stimulate Neuropeptide Y. What is the research on this? There has of course been speculation about the link between diet drinks and metabolic syndrome. —Preceding unsigned comment added by A.k.a. (talk • contribs) 13:42, 11 August 2009 (UTC)

Merger proposal

The article Site-specific functions of Central Neuropeptide Y was recently created, deleted as a copy-vio, and then recreated with a less close paraphrase (although I think it's still too close). Furthermore, the creator of the article appears to have a conflict of interest, as her user name is a shortened version of the names of one of the principal authors the study that was closely paraphrased. I don't know much about the subject material (I saw this originally while patrolling recent changes), but it seems to me that this other article should really be merged in here as it seems to be just a sub-topic of this main topic. Alternatively, the other article needs to be rewritten, so that it's not so close a paraphrase and it's more encyclopedic in tone. Hopefully, editors of this page will have a better understanding than I, and if those here think there's a clear need for a separate article, I'll definitely follow your lead. Qwyrxian (talk) 05:25, 5 April 2011 (UTC)

DEX

DEX is NOT a NPY agonist, that is just absurd. — Preceding unsigned comment added by 129.171.150.141 (talk) 13:54, 3 November 2014 (UTC)