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Antihypertensive response to prolonged tempol in the spontaneously hypertensive rat.

Tempol corrects hypertension without a compensatory sympathoadrenal activation or salt retention and occurs despite increased PRA, accompanied by a reduction in oxidative stress and is maintained during increased salt intake.
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Acute antihypertensive action of nitroxides in the spontaneously hypertensive rat.

The antihypertensive response to piperidine Ns is predicted by SOD mimetic action, and the sensitivity of response is by hydrophilicity.
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Acute antihypertensive action of Tempol in the spontaneously hypertensive rat.

The acute antihypertensive action of Tempol depends on the independent effects of potentiation of nitric oxide and inhibition of the peripheral SNS that involves the activation of K(ATP) channels.
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Abstract 15689: Resistant Rapid Atrial Flutter With Left Atrial Thrombus: A Therapeutic Dilemma

This clinical noninvasive maneuver may be useful and desired in the management of a deteriorating patient with AFL not responsive to chemical rate control and LA thrombus such as the authors' or for an instance when AF in a patient with LA thROMbus was inadvertently converted to SR.
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Primary cilia, A‐kinase anchoring proteins and constitutive activity at the orphan G protein‐coupled receptor GPR161: A tale about a tail

This review introduces GPR161 in the context of Shh signalling and describes the unique features on its C‐terminus, which may influence the function of the receptor, cAMP compartmentalisation and/or trafficking within primary cilia.
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Cardiovascular-Kidney Interactions in Health and Disease Acute antihypertensive action of nitroxides in the spontaneously hypertensive rat

This research presents a novel, scalable, scalable and scalable approach to personalized medicine that aims to improve the quality and quantity of life of patients using a simple, efficient, and scalable method.

ELECTROPHYSIOLOGY

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CONGENITAL ABSENCE OF THE LEFT CIRCUMFLEX ARTERY IN A PATIENT WITH MULTIVESSEL CORONARY ARTERY DISEASE

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Roles of vasoconstrictor prostaglandins, COX-1 and -2, and AT1, AT2, and TP receptors in a rat model of early 2K,1C hypertension.

Whether blood pressure in a rat model of early (3 wk) two-kidney, one-clip Goldblatt hypertension is maintained by AT(1)Rs or AT(2)Rs, driving COX-1 or -2-dependent products that activate TPRs is investigated.
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CALL FOR PAPERS Cardiovascular-Renal Mechanisms in Health and Disease

It is concluded that p22 and NADPH oxidase regulate the SNGFR during ANG II infusion via MD-dependent mechanisms.
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